Excess protein food
Prn favors the disease cancer following mechanisms: increased availability of amines, which coupled with nitrite form carcinogenic nitrosamines, increased consumption of fatty meat ration involves simultaneously increase, as both meat fats are associated with the visible component, but invisible (representing up to 35% the nutritional value of meat) - excess fat in malignant indirect role previously mentioned, provides animal protein tryptophan, which favors the carcinogenic action through its metabolites. The action of intestinal microbial flora on the high availability of amino acids increases the number of food produced with carcinogenic action on the colon, with high-fat diet, excess protein encourages the growth of anaerobic microbial flora developing products related to carcinogens. Do not forget that the thermal treatment processes sometimes form preteinelor pyrolysis products of tryptophan (Trp-P-1 and Trp-P-2) with proven experience carcinogenic properties [2].
His statistics showed that the number of cancer cases in workers in the meat industry is higher than the average of cases in the whole population.
Alcohol
Intervene directly in setting the cancer disease through profound nutritional imbalances that train (there's a deficiency of vitamins and minerals especially role in anticancer protection) by facilitating solvent transport through cell membranes carcinogens by irritant to the tissues. It is possible, however, the coexistence of carcinogens from industrial raw materials or processing.
Fiber Deficiency
Fiber components of plants that are not subject to digestion in the small intestine. Some of them can be degraded in the large intestine. Eaten in small quantities favors colon cancer. He noted that the number of colon cancer in industrialized countries compared to the number of developing countries is much higher. The explanation should be given more marked in part by consumption of dietary fiber in less industrialized countries. It appears that increased fiber intake may actually limit indirect carcinogenic action of other nutrients.
The mechanisms by which low dietary fiber intake promotes colonic cancer are multiple: reduced rate of intestinal transit, increase the share of anaerobic microbial flora, reduce fecal loss of fat and protein. Intestinal transit rate is lower in those with low consumption of dietary fiber, because they occur in increasing fecal volume and intestinal receptor stimulation, resulting in stimulation of fecal evacuation. As transit is slowed, the more contact between carcinogens and the intestinal wall is extended, increasing the risk of malignant degeneration initiation.
Reducing dietary fiber intake in animal fats and proteins favorarea stimulate the creation of a higher density of intestinal anaerobic bacteria in aerobic damage. As mentioned before, increasing the ratio of anaerobic / aerobic favors intestinal malignancy. On the other hand, decreased intake of cellulose products decreases acidity favoring the development of intestinal microbial flora involved in the metabolism of bile acids produced carcinogens.
Reducing consumption of dietary fiber increases the availability of bile acids for intestinal microbial flora (bile acids are not absorbed by the cellulosic substrate and remove the faeces), which can turn them in certain conditions or procancerigene carcinogens. It also was a reduction in fecal excretion of fat and nitrogenous substances, which indirectly promotes the malignant process.
Deficiencies of certain micronutrients
Mechanisms favors carcinogenesis process variables. Thus, iodine deficiency facilitates thyroid cancer, and vitamin B2 deficiency of mouth cancer, larynx and esophagus. Also, vitamin A deficiency promotes formation of gastric cancer, liver cancer pyridoxine deficiency etc..
